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A., Matar, C., Farnworth, E., & Perdign, G

This diet-microbiota-metabolite-immune axis unifies diverse dietary strategies, as each intervention modulates microbial flux toward metabolites that activate conserved anti-tumor pathways (e.g., HDAC inhibition, AhR signaling, STING activation) while mitigating metabolite-driven immunosuppression

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Because PDI bound by 4-HNE was identified in vivo, where GSH is present, it suggests that this compensatory mechanism may be limited by high levels of oxidative stress in which GSH concentrations are low [91]