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however, both the age of onset and disease penetrance vary considerably among different mutations and across endemic regions [6, 7]

The significant increase in pro-inflammatory cytokines in control cows may be attributed to a higher degree of calving stress and inflammatory condition as compared to the micronutrient injected cows which are in accordance with previous reports showing higher levels of pro-inflammatory cytokines in cows suffering from postpartum reproductive problems (4, 5)

hPrdx6-mediated selenocysteine production seems to be independent of the classical pathway that requires selenocysteine lyase (SCLY) for transferring HSe - to SEPHS2 [26]