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If the wild type CFTR is transfected into the cells, the rate of GSH secretion is increased to the level seen in normal cells (Gao et al., 1999)

[] The increased AGT in the septic liver causes cardiac dysfunction by Ang II-dependent (by releasing systemic IL-1) and Ang II-independent mechanisms, in the latter one, AGT is internalized by LRP1 in cardiac fibroblasts, and then, induces NLRP3 inflammasomes-mediated IL-1 release in cardiac fibroblasts, and the released IL-1 further inhibits SERCA2a (sarco[endo]plasmic reticulum Ca 2+ -ATPase 2a) abundances in cardiomyocytes

6 (download XLSX Statistical Source Data Unprocessed western blots Source Data Extended Data Fig

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