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Our data demonstrate that a common mitochondrial uncoupler dinitrophenol, which extends the mouse lifespan 36 , induces tbb-6 expression similarly as does acivicin, further indicating that both chemicals target ETC (Fig

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Of these, arm1, in which residues S17, I18, V21 and A22 were deleted, while still containing G19 and P20 at the -hairpin tip, had no significant effect on the activity of Lgt, that is, colonies were formed normally in the complementation assay (Supplementary Figs 3c and 4a)