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The mechanism formally parallels the -elimination reaction catalyzed by PLP-dependant enzymes which accept cysteine thioethers as substrates and likely play a role in the renal toxicity and carcinogenicity of halogen-alkenes through their urinary metabolites [125]

However, even in ASD individuals without clinical GI symptoms, low level dysregulation of cellular processes such as mitochondrial function would still be present in the gut, which may benefit from trace amine modulation

After confirming that drug-treated 4T1 cells could be efficiently internalized by BMDCs, we investigated whether this process was accompanied by alterations in the functional status of BMDCs