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Further mechanistic studies suggest that the cardioprotective effects of APS may be mediated by the regulation of the PI3K/Akt/mTOR signaling pathway, which is essential for maintaining cardiomyocyte survival, regulating autophagy, and inhibiting apoptosis (Luo et al., 2014)

Moreover, PARP-1 regulates HIF-1 accumulation during nitric oxide and oxidative stress pathways ( Regarding post-transcriptional regulation, the evidence remains largely inferential

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