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These observations suggest that the consumption of cysteine and GSH in the presence of 3MP in cells expressing 3MST is due to the production of Cys-SSH, GSSH, GSSG and their polysulfide counterparts (Cys-SS n H, GSS n H, and GSS n G, n 2)

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The latest research shows that there is a cross - regulation mechanism between ferroptosis and cuproptosis through pathways such as ROS accumulation, mitochondrial damage, and TCA cycle, suggesting that they may coordinately regulate the fate of tumors (Liu and Chen, 2024)