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Importantly, this combination therapy also augments the efficacy of anti-PD-1 antibodies by activating stimulator of interferon genes (STING)-dependent antitumor immunity

Glanzmann thrombasthenia is an autosomal recessive disorder of primary hemostasis caused by a quantitative or qualitative deficiency of the platelet surface receptor complex

Abstract RNA-binding proteins (RBPs) containing low-sequence complexity domains mediate the formation of cellular condensates and membrane-less organelles with biological functions via liquidliquid phase separation (LLPS)

Moreover, we found that cells with mut-p53 accumulation had significantly higher amounts of ROS following APR-246 treatment when compared to cells with low levels or absence of mut-p53 protein (Fig