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TCE exposure, which is known to induce/exacerbate SLE in both experimental animals and humans, is also reported to cause alterations in the gut microbiome with increased abundance of genus Bifidobacterium and bacterial family Enterobacteriaceae as well as lower abundance of the genus Bacteroides and Lactobacillus in MRL+/+ mice at a high but occupationally relevant TCE dose compared to controls (100)

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Upon ligand binding, VEGFR-2 undergoes dimerization and autophosphorylation, initiating downstream signaling cascades that include the phosphoinositide 3-kinase (PI3K)/Akt and the mitogen-activated protein kinase (MAPK)/ERK pathways

[DOI] [PubMed] [PMC] Lee DY, Kim EH