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A literature review published in 1992 reports hepatotoxicity in six patients from unmodified niacin, two patients from sustained release and in 10 patients after changing from unmodified to time-release preparations, likely due to ingesting time-release niacin at doses well above the usual therapeutic doses [12]

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In support of the hypothesized role of glucocorticoid transcriptional regulation of extrahypothalamic GRs in addiction-like behavior, an intra-amygdala infusion of an antisense oligonucleotide that blocked GR transcriptional activity reduced addiction-like behaviors