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Description

T.JensenT.MullerL.PerssonS.et al

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Excessive Toll-like receptor (TLR) activation contributes significantly to IBD pathogenesis, making TLR inhibition a promising therapeutic strategy

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Expression of glucagon-like peptide 1 receptor in a murine C cell line: regulation of calcitonin gene by glucagon-like peptide 1

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Heterologous expression of bacterial natural product biosynthetic pathways

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