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It adds another layer to our understanding of metformin's mechanism of action

The combination hypothesis is that amylin and GLP-1 signaling are complementary rather than redundant

This conservative approach has merit because it keeps a higher dose in reserve as a strategic tool

For example, neurexin-1 and LRRTM4 bind in a Ca 2+ -dependent manner [61], which may suggest that synaptic adhesion molecules can sense synaptic activity (Ca 2+ release) and thereby cause a change in protein structure to form a protein conformation for binding by both pre- and postsynaptic adhesion molecules, leading to the formation of transsynaptic adhesion molecules