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The convergence of evidence from animal models, human cohort studies, and mechanistic investigations strongly supports the conclusion that EED exposure creates heritable reproductive dysfunction through epigenetic mechanisms, fundamentally expanding our understanding of environmental health risks beyond individual exposure effects

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While gastrointestinal AEs such as nausea and vomiting dominate safety reports 8 and recent pharmacovigilance studies highlight increasingly recognized risks including acute pancreatitis, nephritis, hypotension, and syncope 9,10,11 , emerging data suggest that GLP-1 RAs may be associated with neurological adverse events (NAEs) 12,13